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More recently, it was shown that adrenaline-dependent β 2 -AR signaling activation is required for the development of the asthma phenotype in an allergen-driven (Ova S/C) murine asthma model, and chronic treatment with various β -blockers has yielded conflicting results, likely due to drug-specific biased signaling (Thanawala et al., 2013, 2015)

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